Rsv-s8 manual

There is a resonance peak at 8kHz. As such, measurements after this point should not be considered entirely accurate. Oh boy, another Harman-inspired IEM. Now as might be expected, you have your sub-bass oriented curve, flat lower-midrange, and generally neutral upper-midrange. I hear reasonable amounts of slam and texture; decay, as well, seems to stretch out a tad longer than your average BA monitor. I suppose it also bears repeating that this type of midrange tuning is what I perceive as dead-neutral, although it is not necessarily my desired midrange tuning in terms of preference.

Some might find it a tad lean, perhaps dry, with male vocals too. Something that needs to be highlighted, however, is the treble on the RSV. Where the RSV is decidedly not in the realm of the ideal, however, is technicalities. Transient attack has a softness to it not unlike the venerable 64 Audio U12T ; however, the RSV takes a hit on both the fronts of attack incisiveness and decay. Micro-delinations in decay also sound far too smoothed over, thus failing to render trailing, vocal consonances and instrument reverb.

But the way the RSV scales dynamic swings is also somewhat strange. Of course, I have been unrelenting in my critique. Within the context of its price bracket, the RSV is a decent technical performer.

Timbre is certainly better than your average BA monitor; a likely benefit of the smoothing over in note decay. Imaging and layering are generally fine until the RSV hits a more complex track. I would not say that this is a holographic IEM or anything of the sort; staging is slightly above average to my ears.

You get a more relaxed tuning that maintains a slight upper-midrange tilt and an easy-on-the-ears treble response. On the other hand, I think the RSV is tuned just a tad better, particularly when it comes to treble extension.

That in mind, I think fit - and the SA6 has very good fit - would tip the scales for a lot of listeners. Sometimes rapid antigen tests, reverse-transcription—polymerase chain reaction RT-PCR , or viral culture—all done on nasal washings or swabs.

Because antiviral treatment is not typically recommended, a specific laboratory diagnosis is unnecessary for patient management. However, a laboratory diagnosis may facilitate hospital infection control by allowing segregation of children infected with the same virus.

Rapid antigen tests with high sensitivities for RSV and other respiratory viruses are available for use in children; nasal washings or swabs are used. These tests are less sensitive in adults. Molecular diagnostic assays such as RT-PCR have improved sensitivity and are generally available as single or multiplex assays. Corticosteroids and bronchodilators are generally not helpful and are currently not recommended.

Antibiotics are reserved for patients with fever, evidence of pneumonia on chest x-ray, and clinical suspicion of a bacterial coinfection.

Palivizumab monoclonal antibody to RSV is not effective for treatment. Inhaled ribavirin , an antiviral drug with activity against RSV, has marginal efficacy, is potentially toxic to health care practitioners, and is no longer recommended except for infection in severely immunocompromised patients.

Numerous drugs targeting viral fusion, entry, and replication for adults and infants are currently in development and in clinical trials 1 Treatment reference Respiratory syncytial virus and human metapneumovirus infections cause seasonal lower respiratory tract disease, particularly in infants and young children.

Disease may be asymptomatic, mild Infect Dis Ther 10 Suppl 1 —60, Contact precautions eg, hand washing, gloves, isolation are important, particularly in hospitals. Passive prophylaxis with palivizumab decreases the frequency of hospitalization for RSV in high-risk infants.

It is cost-effective only for infants at high risk of hospitalization, including those who. Are 1 year with hemodynamically significant congenital heart disease. Are 1 year with chronic lung disease of prematurity gestational age Are born at 29 weeks gestation and are 1 year old at the start of RSV season.

Have chronic lung disease of prematurity in the 2nd year of life and have received treatment chronic corticosteroid or diuretic treatment or continued need for oxygen therapy within 6 months of RSV season.

Infants in the 1st year of life who have anatomic pulmonary abnormalities that impair the ability to effectively clear the upper airways. Subsequent doses are given at 1-month intervals for the duration of the RSV season usually a total of 5 doses. See also the American Academy of Pediatrics' updated policy statement about palivizumab prophylaxis for infants and young children who are at increased risk of hospitalization for RSV.

Several maternal, pediatric, and adult RSV vaccines are in development in clinical trials 1 Prevention reference Respiratory syncytial virus and human metapneumovirus infections cause seasonal lower respiratory tract disease, particularly in infants and young children. Respiratory syncytial virus RSV and human metapneumovirus usually cause a syndrome of bronchiolitis, but pneumonia may occur. Diagnosis is usually clinical, but testing, including rapid antigen tests and molecular assays eg, reverse-transcription—polymerase chain reaction , is available.

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